Kim Ravnskjaer was appointed Associate Professor at the Department of Biochemistry and Molecular Biology (BMB) in Spring 2018. He earned his PhD-degree from the University of Southern Denmark in 2006 and joined the laboratory of Prof. Marc Montminy at the Salk Institute for Biological Studies in La Jolla (US). At Salk, Kim published several papers on the hormonal regulation and dysregulation of hepatic transcription factors and their target genes in various mouse models of Type 2 diabetes. He returned to the University of Southern Denmark in Spring 2015 when he was appointed Assistant Professor.
At the Department of Biochemistry and Molecular Biology, Kim Ravnskjaer and his team focus on vascular and perivascular cell biology in normal physiology and in cardiometabolic disease. This includes the development of NASH and liver fibrosis as result of of obesity and diabetes. In their studies they explore cellular and molecular mechanisms driving the development and regression of liver fibrosis in human and mice. The aim of their work is the advancement of new methods for early detection and for treatment of advanced cardiometabolic disease as manifested in the liver and beyond.
Kim Ravnskjaer is member of the Danish Diabetes Academy (DDA), the European Association for the Study of Diabetes (EASD), and is co-founder of the The Research Unit for Functional Genomics and Metabolism. He was recently awarded stipends from the European Foundation for the Study of Diabetes (EFDS), the European Commission, and the Danish Council for Independent Research.
In ATLAS 2.0 Kim Ravnskjaer leads Work Package 2 on Molecular and cellular mechanisms of hepatic tissue plasticity in the context of obesity.
